psychosis from weed

It only lasts while they're under the influence, and is extremely rare.

 

 
Not true. Some people that are susceptible to mental illness can experience a full blown psychosis from cannabis during and after the high has diminished. It is extremely rare but it does happen, I've seen it happen to one person personally. 
 
I'd say 99.5% of pot smokers will not experience anything like psychosis, but it does happen. 

 

Case to case basis my friends!

 

 
My little sister hangs with some people who smoke excessively, and who are affected by "hash psychosis" as it's known in Denmark. I've witnessed a normally calm friend throw a fit, shouting incoherently about things we didn't understand.
 
It's one of the major negative points people bring up who are against smoking. 
 
I recommend you do some reading online. Check several sources; don't trust what someone spits out on a forum. People love to act informed. 

 

These are fairly good sources that answer your question. Start reading!
 
Assessing the impact of cannabis use on trends in diagnosed schizophrenia in the United Kingdom from 1996 to 2005.       (abst – 2009)    http://www.ncbi.nlm.nih.gov/pubmed/19560900
 
If cannabis caused schizophrenia-how many cannabis users may need to be prevented in order to prevent one case of schizophrenia? England and Wales calculations.    
(abst - 2009)
http://www.unboundmedicine.com/medline/ebm/record/19832786/full_citation/If_cannabis_caused_schizophrenia_how_many_cannabis_users_may_need_to_be_prevented_in_order_to_prevent_one_case_of_schizophrenia_England_and_Wales_calculations_
 
Minimal Relationship Between Cannabis And Schizophrenia Or Psychosis, Suggested By New Study       (news - 2009)       http://www.sciencedaily.com/releases/2009/10/091022101538.htm
 
New study suggests minimal relationship between cannabis and schizophrenia or psychosis       (news – 2009)             http://www.physorg.com/news175425054.html
 
Science: The development of the number of new schizophrenia cases in the UK does not support the hypothesis that cannabis use increases schizophrenia risk          (news – 2009)
http://www.cannabis-med.org/english/bulletin/ww_en_db_cannabis_artikel.php?id=299
 
THC can improve symptoms of schizophrenia        (news – 2009)
http://www.cannabis-med.org/index.php?tpl=cannabinoids&id=233&lng=en&red=cannabinoidslist
 
Do patients think cannabis causes schizophrenia? - A qualitative study on the causal beliefs of cannabis using patients with schizophrenia        (full - 2010)  http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2954921/?tool=pmcentrez
 
Popular intoxicants: what lessons can be learned from the last 40 years of alcohol and cannabis regulation?       (abst – 2011)        http://www.ncbi.nlm.nih.gov/pubmed/21926420
 
 
Marijuana Compound May Beat Antipsychotics at Treating Schizophrenia
(news – 2012)    
 http://psychcentral.com/news/2012/06/07/marijuana-compound-may-beat-antipsychotics-at-treating-schizophrenia/39803.html
 
Marijuana Use Linked to Better Adherence in Psychosis          (news – 2012)
http://www.clinicalpsychiatrynews.com/index.php?id=2407&cHash=071010&tx_ttnews[tt_news]=138709
 
 
You can read more in "Granny Storm Crow's List"!  To get your free coipy, check out the bottom of my sig. If you are on an app that does not show sigs, you can just PM me your email and I'll get a copy off to you today!
 
 
Granny

 

 
It seems like it is more to do with which cannabinoid rather than a Sativa / Indica thing.  If the answer is not in the list Granny just posted it should be in the full list she offered.
 
The with or without weed question is not yet fully answered.  They are still having trouble understating just what is going wrong in schizophrenia.  In order to answer the question they will have to know what percentage of the at risk population develops and by default fails to develop active disease among non-cannabis users and then compare the result with the same result from the cannabis using population.
 
Your question made me curious, this looks interesting......
I think I'll read it tonight, care to join me?
 
How Genes and Environmental Factors Determine the Different Neurodevelopmental Trajectories of Schizophrenia and Bipolar Disorder
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3283142/?report=classic
 
 - Edit -
This looks interesting too:
Effects of Cannabis Use on Human Brain Structure in Psychosis: A Systematic Review Combining In Vivo Structural Neuroimaging and Post Mortem Studies
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3474956/?report=classic
 
 - Edit 2 -
The 2nd paper creates a interesting paradox.  Independently we know that cannabis appears to be protective to the nervous system in M.S. We also know that cancer patients count on cannabinoids causing apoptosis in many cancers.  Do you suppose that the loss of brain volumes in Psychosis is revealing a defect in the Apoptosis mechanism in the brain?  We know the disease progresses independently of cannabis use and that brain volumes decrease in non cannabis users also.  It could suggest that endocannabinoids are also causing tissue loss......

 

Apologies for derailing the topic, but @Honokiol have you seen this? http://www.washington.edu/news/2013/12/12/scientists-discover-double-meaning-in-genetic-code/
 
With so many illnesses in general having a genetic component I will be sure to follow these studies in the future. I wasn't sure if anyone else had seen the article or if there would be interest.
 
If anyone enjoys reading fiction and topics focusing on mental illness then Wally Lamb's "I Know This Much Is True" may interest you. It is a long narrative about twin brothers and one has paranoid schizophrenia. The book is told from the unaffected brother's POV and is very interesting. I read it straight through without pausing for sleep or anything else when I was in high school. Discovered the book and curled up in my closet and never came out until I was finished reading. 
 

 

That is cool
 

 
That is cool!
 
I spend most of my time studying the "Family Illnesses" in my family but with reading research papers I run across interesting side bits of information, "By-Catch".  For my blood relations it is mostly auto-immune disease.   The big confounder I'm looking at with genetics is diet.  Diet can change both direct expression of existing genetic code and it is passed down through generations like genetic code.  Mom teaches the kids what to eat & how to cook.  Her diet along with Dad's prior to conception will effect the offspring as well as her diet during pregnancy.

 

If someone is predisposed to mental illness, pot can cause it to permenately manifest.
Also, some people with mental illness use recreational drugs. The question is did the recreational drug use cause the illness or is it people with mental illness self-medicate with recreational drugs. We don't know.

 

 
For me the answer appears to be a blend of both.  I had GERD and OSA before either had a diagnostic code or treatment.  I was self medicating a health problem, a "sleep disorder" I had on clue I had.  My Dr. in the days when I first started complaining about the symptoms hat no clue about it either nor any training in the treatment of it.
 
Who is to say that the "recreational users" among us do not have a medical condition that they and their Dr's have no clue about today.  The fact that our medicine makes them feel better is good enough for me for now.
 
Clinical Endocannabinoid Deficiency has been put forward as a partial root cause of Auto-Immune diseases as a group.  It is emerging from medical research slowly as we speak.  Depression appears to be part of this group also.   The 2nd best antidepressant I've had in my life is Cannabis which is one of the only two I can tolerate. Cannabis also appears useful in relieving symptoms of most auto immune diseases.  The number one best treatment in my life for depression has been Omega-3 fats which has only recently come to light as a source of endocannabinoids.
 
The patients we are talking about in this thread already have their disease long before they become symptomatic.  The fact that Cannabis can cause the patient to become symptomatic at a younger age is tragic but does not change what the outcome of the disease will be.  If plant cannabinoids are causing apoptosis in genetically abnormal brain cells and the disease can progress with or without plant cannabinoids then endocannabinoids must also be causing the same apoptosis even though it appears to occur at a slower pace.  There is an up side though because it may be revealing the mechanism of what is occurring and that can lead to a better outcome for future patients who have not yet developed symptoms today.

 

It is literally impossible for a cannabinoid to cause psychosis.

 

Literally impossible? You're joking, right? If not, what is the basis for that statement?

 

\tthis was in the NORML news letter this week...\t \t \tStudy: Cannabis Use Unlikely To Cause SchizophreniaBoston, MA: Family genetics, rather than adolescent cannabis use, increases risk of schizophrenia, according to a case controlstudy published online in the journalSchizophrenia Research.
Investigators from Harvard Medical School compared the family histories of 108 schizophrenia patients and 171 individuals without schizophrenia to assess whether youth cannabis consumption was an independent factor in developing the disorder. Researchers reported that a family history of schizophrenia increased the risk of developing the disease, regardless of whether or not subjects consumed cannabis as adolescents.
"There was an increased morbid risk for schizophrenia in relatives of the cannabis using and non-using patient samples compared with their respective non-psychotic control samples," authors reported. "There was no significant difference in morbid risk for schizophrenia between relatives of the patients who use or do not use cannabis."
They concluded: "The results of the current study, both when analyzed using morbid risk and family frequency calculations, suggest that having an increased familial risk for schizophrenia is the underlying basis for schizophrenia in these samples and not the cannabis use. While cannabis may have an effect on the age of onset of schizophrenia it is unlikely to be the cause of illness."
Previous studies have reported that rates of schizophrenia have remained steady for decades despite changing patterns in cannabis use among the population.
Commenting on the study's findings, NORML Deputy Director Paul Armentano said: "Concerns regarding cannabis' potential impact on psychiatric illnesses arguably warrant further study. However, such concerns - even if confirmed - are not persuasive justifications for continuing cannabis criminalization. Just the opposite is true. There are numerous adverse health consequences associated with alcohol, tobacco and prescription drugs, all of which are far more dangerous and costlier to society than cannabis. It is precisely because of these consequences that these products are legally regulated and their consumption is restricted to specified consumers and settings. This same principle ought to apply to cannabis."
For more information, please contact Paul Armentano, NORML Deputy Director, at: <a data-ipb='nomediaparse' href='mailtoaul@[member="NORML"].org'>paul@[member="NORML"].org</a>. Full text of the study, "A controlled family study of cannabis users with and without psychosis," appears in Neuropsychopharmacology.

\tStudy: Cannabidiol Holds Promise For Treating Psychosis<div>Utrecht, The Netherlands: Cananbidiol(CBD), a non-psychotropic cannabinoid, alleviates psychotic symptoms and may hold promise as an alternative antipsychotic treatment, according to a review published in the November issue of the journalNeuropsychopharmacology.
Investigators in the Netherlands and in the United Kingdom reviewed preclinical and clinical data on the use of CBD as an antipsychotic agent. Authors reported that both animal and human studies document the ability of CBD to mitigate symptoms of psychosis. Specifically, CBD administration is associated with improved symptoms in clinical evaluations of patients with schizophrenia, Parkinson's disease, and ketamine-induced dissociative and psychotic symptoms.
Investigators also highlighted a 2012 double-blind, randomized placebo-controlled trial assessing CBD versus the prescription anti-psychotic drugamisulpride in 42 subjects with schizophrenia and acute paranoia. Authors reported that both CBD and the prescription drug were associated with "equally significant clinical improvement" in this patient population, but that cannabidiol "possessed significantly less side effects."
They concluded: "[E]vidence from several study domains suggests that CBD has some potential as an antipsychotic treatment. ... Given the high tolerability and superior cost-effectiveness, CBD may prove to be an attractive alternative to current antipsychotic treatment."
Previous human trials assessing the administration of CBD in healthy human subjects report that the cannabinoid is "safe and well tolerated."
Separate investigations of CBD, primarily in animal models, havedocumented the cannabinoid to possess a variety of therapeutic qualities, including anti-inflammatory, anti-diabetic, anti-epileptic, anti-cancer, and bone-stimulating properties. Recently, the FDA approved the experimental use of CBD extracts for the treatment of a rare form of intractable pediatric epilepsy known as Dravet syndrome. Preliminary clinical trials assessing the safety and tolerability of the compound in children are scheduled to begin imminently.
For more information, please contact Paul Armentano, NORML Deputy Director, at: <a data-ipb='nomediaparse' href='mailtoaul@[member="NORML"].org'>paul@[member="NORML"].org</a>. Full text of the study, "Cannabidiol as a potential treatment for psychosis," appears in Neuropsychopharmacology.


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myth was debunked...yet the noob accounts keep popping in to support it...hmm...cointelpro tactics anyone?

 

 
I did not say Cannabis caused Psychosis.
 
I said cannabinoids incl. endocannabinoids are participating in the pathology by causing apoptosis in genetically damaged brain cells for the same reason it cures cancer.  The genetic damage is already present and which source of cannabinoid or endocannabinoid is irrelevant.  It is the preexisting genetic damage that is predisposing the cells to the apoptic mechanism that will progress through endocannabinoid activity whether cannabis is used or not.
 
This is in agreement with the Normal statement that Cannabis is not the cause.  That does not mean it does not participate in the progression and or acceleration of the disease, just that it is not the source of the disease.

 

yup...just throwing in my 2 cents.....
 
 
thought this was interesting
 
"While cannabis may have an effect on the age of onset of schizophrenia it is unlikely to be the cause of illness."

 

 
Sorry I misunderstood.
 
This mechanism could explain some of the argument that began in the 1800's particularly in Egypt but also in India where at least part of the "Causes Insanity" argument originated.  This is among the older letters in the discussion, I'm having trouble finding the top of the thread.
 
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2422352/pdf/brmedj08898-0037b.pdf
 
See also:
http://www.ncbi.nlm.nih.gov/pmc/?term=%22Indian+Hemp%22+%22Egypt%22+%221893%22
 
It was quite the flame war for it's day.

 

 
I hate my fellow Danes so much sometimes, it's about the only fucking argument they (they = the sheeple) use here, it makes me want to throw up all over whoever says it so badly, it actually happens sometimes. Alone the fact that Danes still only discuss cannabis as hash is completely laughable.